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Actions of AAS to inhibit activity of the dynorphin/kappa opioid receptor system in the nucleus accumbens are particularly intriguing. Treatment with kappa receptor antagonists in the nucleus accumbens produces anxiolytic and antidepressant effects (81. The AAS-induced reduction in nucleus accumbens dynorphin might also facilitate dopaminergic activity (78 ). However it is notable that, unlike many other drugs of abuse, AAS do not acutely stimulate dopamine release in the nucleus accumbens (84 ). This is consistent with the relatively slow time-course of AAS action, and may account for the absence of acute intoxicating effects. Studies using the opioid antagonist naloxone have yielded variable results in AAS-treated animals. Nandrolone pretreatment enhanced withdrawal symptoms to naloxone in morphine-dependent mice (80 ), and naloxone reversed testosterone-induced locomotor depression in hamsters (75 ).


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